Estradiol attenuates programmed cell death after stroke-like injury.

Are estrogens protective or risk factors in brain injury and neurodegeneration? Reevaluation after the Women's health initiative.

Stroke injury in rats causes an increase in activin A gene expression which is unaffected by oestradiol treatment.

Improved ceramic-based multisite microelectrode for rapid measurements of L-glutamate in the CNS.

Real time in vivo measures of L-glutamate in the rat central nervous system using ceramic-based multisite microelectrode arrays.

Microelectrode array studies of basal and potassium-evoked release of L-glutamate in the anesthetized rat brain.

Differential modulation of estrogen receptors (ERs) in ischemic brain injury: a role for ERalpha in estradiol-mediated protection against delayed cell death.

Differential modulation of estrogen receptors (ERs) in ischemic brain injury: a role for ERalpha in estradiol-mediated protection against delayed cell death.

[Anonymous].  2006.  Differential modulation of estrogen receptors (ERs) in ischemic brain injury: a role for ERalpha in estradiol-mediated protection against delayed cell death.. Endocrinology. 147(6):3076-84.

A case of paraneoplastic myelopathy associated with the neuromyelitis optica antibody.

Amperometric measures of age-related changes in glutamate regulation in the cortex of rhesus monkeys.

Collagen VI protects neurons against Abeta toxicity.