Novel calcium-related targets of insulin in hippocampal neurons.

FK506-Binding Protein 12.6/1b, a Negative Regulator of Ca<sup>2+</sup> , Rescues Memory and Restores Genomic Regulation in the Hippocampus of Aging Rats.

FK506-Binding Protein 12.6/1b, a Negative Regulator of Ca<sup>2+</sup> , Rescues Memory and Restores Genomic Regulation in the Hippocampus of Aging Rats.

[Anonymous].  2018.  FK506-Binding Protein 12.6/1b, a Negative Regulator of Ca2+ , Rescues Memory and Restores Genomic Regulation in the Hippocampus of Aging Rats.. The Journal of neuroscience : the official journal of the Society for Neuroscience. 38(4):1030-1041.

Expression of a Constitutively Active Human Insulin Receptor in Hippocampal Neurons Does Not Alter VGCC Currents.

Insulin Receptor Plasma Membrane Levels Increased by the Progesterone Receptor Membrane Component 1.

Aging-Related Calcium Dysregulation in Rat Entorhinal Neurons Homologous with the Human Entorhinal Neurons in which Alzheimer's Disease Neurofibrillary Tangles First Appear.

Aging-Related Calcium Dysregulation in Rat Entorhinal Neurons Homologous with the Human Entorhinal Neurons in which Alzheimer's Disease Neurofibrillary Tangles First Appear.

[Anonymous].  2018.  Aging-Related Calcium Dysregulation in Rat Entorhinal Neurons Homologous with the Human Entorhinal Neurons in which Alzheimer's Disease Neurofibrillary Tangles First Appear.. Journal of Alzheimer's disease : JAD.

Inclusion-body myositis and Alzheimer disease: two sides of the same coin, or different currencies altogether?

Beta-amyloid mediated nitration of manganese superoxide dismutase: implication for oxidative stress in a APPNLH/NLH X PS-1P264L/P264L double knock-in mouse model of Alzheimer's disease.

Beta-amyloid mediated nitration of manganese superoxide dismutase: implication for oxidative stress in a APPNLH/NLH X PS-1P264L/P264L double knock-in mouse model of Alzheimer's disease.

[Anonymous].  2006.  Beta-amyloid mediated nitration of manganese superoxide dismutase: implication for oxidative stress in a APPNLH/NLH X PS-1P264L/P264L double knock-in mouse model of Alzheimer's disease.. The American journal of pathology. 168(5):1608-18.

Abeta solubility and deposition during AD progression and in APPxPS-1 knock-in mice.

In vitro and in vivo degradation of Abeta peptide by peptidases coupled to erythrocytes.

Peripherally expressed neprilysin reduces brain amyloid burden: a novel approach for treating Alzheimer's disease.