The p38 MAP Kinase Family as Regulators of Proinflammatory Cytokine Production in Degenerative Diseases of the CNS.

Oxidative Stress and the Aging Brain: From Theory to Prevention

CX3CL1 reduces neurotoxicity and microglial activation in a rat model of Parkinson's disease.

Microglial p38α MAPK is a key regulator of proinflammatory cytokine up-regulation induced by toll-like receptor (TLR) ligands or beta-amyloid (Aβ).

Microglial p38α MAPK is a key regulator of proinflammatory cytokine up-regulation induced by toll-like receptor (TLR) ligands or beta-amyloid (Aβ).

[Anonymous].  2011.  Microglial p38α MAPK is a key regulator of proinflammatory cytokine up-regulation induced by toll-like receptor (TLR) ligands or beta-amyloid (Aβ).. Journal of neuroinflammation. 8:79.

Neuron-Microglia Dialogue and Hippocampal Neurogenesis in the Aged Brain.

CX3CR1 deficiency leads to impairment of hippocampal cognitive function and synaptic plasticity.

Microglial p38α MAPK is critical for LPS-induced neuron degeneration, through a mechanism involving TNFα.

Early stage drug treatment that normalizes proinflammatory cytokine production attenuates synaptic dysfunction in a mouse model that exhibits age-dependent progression of Alzheimer's disease-related pathology.

Early stage drug treatment that normalizes proinflammatory cytokine production attenuates synaptic dysfunction in a mouse model that exhibits age-dependent progression of Alzheimer's disease-related pathology.

[Anonymous].  2012.  Early stage drug treatment that normalizes proinflammatory cytokine production attenuates synaptic dysfunction in a mouse model that exhibits age-dependent progression of Alzheimer's disease-related pathology.. The Journal of neuroscience : the official journal of the Society for Neuroscience. 32(30):10201-10.

Targeting astrocytes ameliorates neurologic changes in a mouse model of Alzheimer's disease.

Deficiency in p38β MAPK fails to inhibit cytokine production or protect neurons against inflammatory insult in in vitro and in vivo mouse models.

Deficiency in p38β MAPK fails to inhibit cytokine production or protect neurons against inflammatory insult in in vitro and in vivo mouse models.

[Anonymous].  2013.  Deficiency in p38β MAPK fails to inhibit cytokine production or protect neurons against inflammatory insult in in vitro and in vivo mouse models.. PloS one. 8(2):e56852.